邮箱：xxjiang@fudan.edu.cn
研究方向:代谢性疾病及神经功能紊乱的机制
经历简介：
教育经历：
2015.9-2020.6 中国科学院上海营养与健康研究所 生理学 博士
2011.9-2015.6 河南理工大学生物技术学士
工作经历：
2020.7-2022.7 中国科学院上海营养与健康研究所 博士后
2023.1至今 复旦大学脑科学转化研究院 博士后
代表性文章
1.Jiang X, Liu K, Jiang H, Yin H, Wang ED, Cheng H, Yuan F, Xiao F, Wang F, Lu W, Peng B, Shu Y, Li X, Chen S, Guo F. SLC7A14 imports GABA to lysosomes and impairs hepatic insulin sensitivity via inhibiting mTORC2. Cell Rep. 2023 Jan 31;42(1):111984.
https://doi.org/10.1016/j.celrep.2022.111984
2.Jiang X, Xiao F, Guo F. Oocyte TET3: an epigenetic modifier responsible for maternal inheritance of glucose intolerance. Signal Transduct Target Ther. 2022 Oct 8;7(1):357.
https://doi.org/10.1038/s41392-022-01170-0
3. Yin H, Yuan F, Jiao F, Niu Y,Jiang X, Deng J, Guo Y, Chen S, Zhai Q, Hu C, Li Y, Guo F. Intermittent Leucine Deprivation Produces Long-lasting Improvement in Insulin Sensitivity by Increasing Hepatic Gcn2 Expression. Diabetes. 2022 Feb 1;71(2):206-218.
https://doi.org/10.2337/db21-0336
3. Jiang X, Guo F. [The role and mechanism of amino acids in regulating hepatic glucose and lipid metabolism]. Sheng Li Xue Bao. 2021 Oct 25;73(5):723-733.
https://doi.org/10.13294/j.aps.2021.0064
4. Yuan F, Jiang H, Yin H,Jiang X, Jiao F, Chen S, Ying H, Chen Y, Zhai Q, Guo F. Activation of GCN2/ATF4 signals in amygdalar PKC-δ neurons promotes WAT browning under leucine deprivation. Nat Commun. 2020 Jun 5;11(1):2847.
https://doi.org/10.1038/s41467-020-16662-2
5. Deng J, Guo Y, Yuan F, Chen S, Yin H,Jiang X, Jiao F, Wang F, Ji H, Hu G, Ying H, Chen Y, Zhai Q, Xiao F, Guo F. Autophagy inhibition prevents glucocorticoid-increased adiposity via suppressing BAT whitening. Autophagy. 2020 Mar;16(3):451-465.
https://doi.org/10.1080/15548627.2019.1628537
6. Deng Y, Xiao Y, Yuan F, Liu Y,Jiang X, Deng J, Fejes-Toth G, Naray-Fejes-Toth A, Chen S, Chen Y, Ying H, Zhai Q, Shu Y, Guo F. SGK1/FOXO3 Signaling in Hypothalamic POMC Neurons Mediates Glucocorticoid-Increased Adiposity. Diabetes. 2018 Apr;67(4):569-580.
https://doi.org/10.2337/db17-1069
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邮编：200032 
电话/传真：021-54237056 
邮箱：itbr@fudan.edu.cn 
